Insulin sensitivity function

«Insulin resistance (IR) is considered as a pathological condition in which cell fail to respond normally to the hormone insulin.[1] To prevent hyperglycemia and noticeable organ damage over time,[2] the body produces insulin when glucose starts to be released into the bloodstream, primarily from the digestion of carbohydrate in the diet. Under normal conditions of insulin reactivity, this insulin response triggers glucose being taken into body cell, to be used for energy, and inhibits the body from using fat for energy, thereby causing the concentration of glucose in the blood to decrease as a result, staying within the normal range even when a large amount of carbohydrate is consumed. Carbohydrate comprise simple sugar, i.e. monosaccharides, such as glucose and fructose, disaccharides, such as cane sugar, and polysaccharides, e.g. starches. Fructose, which is metabolised into triglycerides in the liver, stimulates insulin production through another mechanism, and can have a more potent effect than other carbohydrate. A habitually high intake of carbohydrate, and particularly fructose, e.g. with sweetened beverages, contributes to insulin resistance and has been linked to weight gain and obesity.[3][4][5] If excess blood sugar is not sufficiently absorbed by cell even in the presence of insulin, the increase in the level of blood sugar can result in the classic hyperglycemic triad of polyphagia (increased appetite), polydipsia (increased thirst), and polyuria (increased urination). Avoiding carbohydrate and sugar, a no-carbohydrate diet or fasting can reverse insulin resistance.[6][7]» (wikipedia)

  • Has Subnodes:

Pathways of Insulin sensitivity

Textual paths of Biolinks with Validity Score > 3.0
Uses in-app credits Help
This tool shows paths like: A B C Insulin sensitivity


Visualisation of logical biolinks between drugs, supplements, symptoms etc..